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Here, we analyzed CNV lesion numbers and sizes in a large number of agematched (6weekold) mice and could show that targeting Nlrp3 or Il1r1 significantly reduced CNV lesion numbers (but not CNV lesion sizes), while targeting Il18 did not show a statistically significant change in either CNV lesion numbers or their size (Fig 8A and B, and Appendix Fig S5A and B)

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Next, the study showed that inhibition of Sat1 alleviated MIRI by suppressing ferroptosis in vivo and in vitro , and over-expression of Sat1 promoted MIRI via activation of ferroptosis